Fes

F flat

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Aliase for FES gene

Tirosine proteine kinase, acting upstream of cellular survival recorders and playing a part in the actine skeleton control, the microtubules arrangement, cellular attachments and cellular proliferation. Played a part in FCER1 (high affinity immune globulin egpsilon receptor) mediated signal transmission in mastocytes. Operates upstream of the active FCER1 and fattening stalk cancer KIT proliferation record.

It has a part to play in the control of the granulation of mast cells. Acts as a regulator of cellular differencing and stimulates the growth of neurites in reaction to NGF signals. Significant factor in cellular dispersion and cellular migrations in reaction to HGF-induced ECR induction. It phosphorylates BCR and controls the BCR quinase activities.

IPDnew, Ensembl, Envcode, genetic target for GeneHancer: NENCODE: IPDnew, FRANTOM5, Ensembl, Envcode, genetic target for GeneHancer: NENCODE: IPDnew, FRANTOM5, Ensembl, Envcode, genetic target for GeneHancer: NENCODE: and GeneHancer genetic targets: NENCODE: and GeneHancer genetic targets: NENCODE: Unlike oncoprotein viruses, the quinase activities of FSP/FES are strongly controlled and the quinase is inert in ordinary cell without stimulating agents (PubMed:15485904).

Spiral coils are important for the control of quinase activities. Tirosine proteine kinase, acting upstream of cellular receptor surfaces, which is involved in the actine skeleton control, microtubular arrangement, cellular attachments and cellular proliferation. Played a part in FCER1 (high affinity immune globulin receptor epsilon) mediated signal transmission in mastocytes.

Operates downstream of the active FCER1 and fattening stalk cells and KITs. It has a part to play in the control of the granulation of mast cells. Contributes to the modulation of cellular differencing and stimulates the proliferation of neurites in reaction to NGF signals. Significant contribution to cellular dispersion and cellular migrations in reaction to HGF-induced ECR induction.

Phosphorylizes BCR and controls the BCR quinase activities. Quinase activities are strictly controlled. Activates in reaction to a signal from a cellular interface receptor. No. Zellmembran; periphery membraneprotein; cytoplasmatic side. cytogenesis, focused adhesions. Microtubule associations require quinase activations to be enabled. Enrolled on the side membranes of polarised epoxylic stem cell membranes by interacting with phosphorylated ER.

Detection of tube-shaped membranes in the zytoplasm and at the cytophery. Synonyms : IDI1_002118, KBio2_007405, Bio-E-Gel, Prestwick3_000441, BIDD:PXR0065, 3,17-bêta-Östradiol, Stradiol Synonyms : IDI1_002118, KBio2_007405, Bio-E-Gel, Prestwick3_000441, BIDD:PXR0065, 3,17-bêta-Östradiol, Stradiol, CID5757, Sandrena 1, Estrasorb, nchembio794-comp6, KBio3_000769, 17,bêta-beta. Eestradiol, 17-beta, Estraderm TTS 100, Ginosedol, constituent of Menrium, 17-E, D00105,[2,4,6,7-3H]-E2, NSC-9895, Femtran, Estrogel, Sandrena Gel, Polyestradiol, KBio2_002973, Estrofem Forte,[3H]-estradiol, Femanest,

Beta oestradiol, KBioGR_000405, NCGC00091544-04, Climara Forte, Bio1_000892, E0025, EMS2090E18, dimenformon, BSPBio_001065, Estraderm, SMP1_000121, 17beta oestradiol, oestradiol R, SPBio_002421, ß. 168-comp3, Estrofem 2, Estreva, Spectrum5_002055, Î2. -Eestradiol, 17-.BETA. Eestradiol, Innofem (TN), Extrasorb, Vagifem (TN), Oestroglandol, NCGC00091544-01, Estradiolum[INN], Ovastevol, Beta-estradiol, 17beta-estradiol, 17b-estradiol, Profoliol B, Ovahormon, Estrogel (TN), 17E, E1024_SIGMA, E1132_SIGMA, Oestradiolum, BIDD:

  • Oestradiol, estradiol[USAN: INN ], Dihydroxyesterin, AC-10460, Divigel, 17-beta-OH-estradiol, Estradiol-17 ß, Gynergon, NCGC00091544-00, Estrasorb (TN), Prestwick_207, Estraderm MX, CCRIS 280, Dihydrofolliculin, 1qku, (17?)-estra-1, 3,5(10)-tria-3,17-diol, 17?-estra-1,3,5(10)-tria-3,17-diol, 17?-estradiol, 17?-estradiol, beta-estradiol, esstradiol, esterdiol-17beta, (17beta)-estra-1,3,5(10)-tria-3,17-diol, 17beta oestradiol, oestradiol di( (1,3,5(10)-trien-3,17b-diol, estrogel HBF, oestra-1,3,5(10)-trien-3,17b-diol, profoliol b, progynone, oestradiol hemi-hydrate, (17 alpha)-isomer, oestradiol, (-)-isomer, oestradiol, monoatriumsalt, 17 beta-oestradiol, oestradiol 17 beta, estradiol-monohydrate, estradiol-orion label, oestradiol, (17-alpha)-isomer, oestradiol, soda solution, 17 beta-estradiol,

Eestradiol hemi-hydrate, esstradiol, (+-)-isomer, esstradiol, (16 alfa,17 alfa )-isomer, estradiol-17 alfa, orienton mark OF esradiol, 17 beta-estradiol, esstradiol 17 alfa, esstradiol 17beta, esstradiolhydrous, anhydric, and anhydric, oestradiol, (16 alfa,17 beta)-isomer, oestradiol, (8 alfa,17-beta) -(+-)-isomer, oestradiol, (8 alfa,17 beta)-isomer, oestradiol, (9 beta,17 -isomer, oestradiol, (9 beta,17 beta)-isomer, (9 beta,17 beta)-isomer, publishchem IDs: 7alpha- [9[(4,4,5,5,5,5,5-pentafluoropentyl)sulfinyl]nonyl]-estra-1,3,5(10)-trien-3, 17 betasiol, (7alpha,17beta)-7-{9-[(4,4,5,5,5,5-pentafluoropentyl)sulfinyl]nonyl}estra-1,

FSP/FES supresses anchor-independent regrowth in colorectal carcinoma cells (PubMed:16455651).

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